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The expression of the pro-inflammatory cytokines was significantly retarded in the caftaric acid-treated group, along with restoration of the levels of glutathione and superoxide dismutase

76 in which five out of nine patients with previous progression responded to a 4-week KD with partial remission or stable disease as judged by FDG-PET scans

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c-Myc transcriptionally activates CPT1A to form a mutually reinforcing intermolecular loop in lung cancer cells Our investigation revealed a tight regulation of CPT1A expression by c-Myc (Fig